MOTS-c Co

MOTS-c versus NAD+ and the mitochondrial peptide family

Every row cites its source. Where a cell reflects our own reading of the evidence rather than an external source, the row says so.

MOTS-c is marketed alongside other mitochondrial compounds. The comparison is useful mainly because each one fails a different evidence test, and knowing which failure applies changes what a buyer should ask.

MOTS-c versus NAD+ and the mitochondrial peptide family
DimensionOptionWhat it isGapStatusSister UrlSister NoteSource
MOTS-cMitochondrial-derived peptide encoded in MT-RNR1, injectedRich animal and human-genetics literature, zero published human dosing studiesNot approved; FDA identified no human exposure data; proposed for exclusion from the 503A list--source
Humanin and SHLP1-6The other seven mitochondrial-derived peptides, encoded in the 16S rRNA geneSame family, same preclinical-heavy pattern; humanin rose after exercise in the human study where MOTS-c only trendedNot approved--source
NAD+ (sister site)Coenzyme given by IV drip, shot or capsuleIts problem is molecular substitution: the credible randomized trials tested precursors such as NR and NMN, not infused NAD+Not approved; compounded under an interim FDA policyhttps://nadplusrx.vercel.appOur NAD+ reference site is currently served from a vercel.app address rather than a custom domain.source
CB4211A MOTS-c analog developed by CohBarThe only completed human program in this space, and it published nothingInvestigational; never approved--source
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